Abdominal eggshell calcifications in the newborn: meconium peritonitis
Ipek Guney Varal, Pelin Dogan
The Pan African Medical Journal. ;31:84. doi:10.11604/pamj..31.84.16592

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Images in clinical medicine

Abdominal eggshell calcifications in the newborn: meconium peritonitis

Ipek Guney Varal, Pelin Dogan
Pan Afr Med J. 2018; 31:84. doi:10.11604/pamj.2018.31.84.16592. Published 04 Oct 2018



Meconium peritonitis is a sterile chemical peritonitis caused by intestinal perforation in-utero or shortly after birth and perforation usually occurs due to intrauterine obstruction. Its incidence is approximately 1 in 35,000 births and the mortality was reported to be 60-80%. The radiographic findings of meconium peritonitis are pneumoperitoneum, intestinal obstruction and abdominal calcifications. The classic eggshell calcification occurs owing to the defensive mechanism of the body against the inflammation in an effort to surround the meconium in the abdominal cavity. In a newborn with abdominal distension, meconium peritonitis should be kept in mind, especially if calcified foci are seen on plain abdominal radiographs, since timely diagnosis and surgery improves the survival rates. We present the case of a female newborn of 34 weeks' gestational age was admitted to the neonatal intensive care unit (NICU) with marked abdominal distension. The pregnancy was complicated by maternal polyhydramnios and a cesarean section was performed due to fetal distress. The infant was noted to have a grossly distended abdomen; however, the physical examination was otherwise unremarkable. The abdominal radiograph revealed distension of the bowel loops and free air with multiple peritoneal calcifications (Figure 1). Surgery, performed on day 1 of life, demonstrated ileal atresia and perforation in the distal part of the ileum. Solid meconium, which leaked into the abdominal cavity was removed and the atresic bowel was resected and anastomosed. The infant regained bowel function on postoperative day 7 and discharged home on the third week after gradual advancement to full enteral feeding.


Corresponding author:
Ipek Gney Varal, Department of Pediatrics, Division of Neonatology, University of Health Sciences, Bursa Yksek Ihtisas Teaching Hospital, Bursa, Turkey
ipekguneyvaral@gmail.com

©Ipek Guney Varal et al. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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