Air crescent sign: typical case of invasive pulmonary aspergillosis
Mrinmayee Vijay Mayekar, Neha Phate
Corresponding author: Mrinmayee Vijay Mayekar, Department of Respiratory Medicine, Datta Meghe Institute of Higher Education and Research, Wardha, Maharashtra, India
Received: 24 Jan 2023 - Accepted: 25 Feb 2023 - Published: 15 Mar 2023
Domain: Pulmonology
Keywords: Invasive aspergillosis, air crescent, post tubercular
©Mrinmayee Vijay Mayekar et al. Pan African Medical Journal (ISSN: 1937-8688). This is an Open Access article distributed under the terms of the Creative Commons Attribution International 4.0 License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Cite this article: Mrinmayee Vijay Mayekar et al. Air crescent sign: typical case of invasive pulmonary aspergillosis. Pan African Medical Journal. 2023;44:130. [doi: 10.11604/pamj.2023.44.130.39058]
Available online at: https://www.panafrican-med-journal.com//content/article/44/130/full
Air crescent sign: typical case of invasive pulmonary aspergillosis
&Corresponding author
A 56-year-old male patient presented with complaints of dyspnoea on exercise for the last two months, recurrent hemoptysis for the past two months, and cough with expectoration for the past three months. In addition to this, over the past two months he has been losing weight and his hunger. He had a history of pulmonary Koch´s three years ago, for which he was treated with antitubercular drugs for a period of time equalling six months. In light of the patient's persistent complaints, we decided to perform a chest X-ray on him, followed through a high-resolution computed tomography (HRCT) of his thorax. The outcomes of these investigations hinted to the presence of post-tubercular sequelae in the form of invasive aspergillosis exhibiting an air crescent sign in the patient's left upper lobe. The patient is a well-documented instance of uncontrolled type II diabetes mellitus, which lends credence to our diagnosis.
Figure 1: a large thick wall cavitary lesion with air filled surrounding the devitalized parenchyma in left upper lobe with bilateral emphysematous changes